Media release

From:
JAMA

Prevalence, Characteristics, and Genetic Architecture of Avoidant/Restrictive Food Intake Phenotypes
JAMA Pediatrics

About The Study: This cohort study found that the prevalence of avoidant/restrictive food intake in the general pediatric population was substantial, and affected children had an associated elevated risk of developmental difficulties across multiple domains. The findings suggest a need for broad support interventions and advance understanding of the genetic underpinnings of avoidant/restrictive food intake.

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Research


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Journal/
conference:
JAMA Pediatrics


Organisation/s:
Curtin University, The University of Western Australia, University of Oslo, Norway


Funder:
Drs Bjørndal, Hannigan, Ask,
and Havdahl are supported by funding from the
Norwegian South-East Regional Health Authority
(Helse Sør-Øst; grant 2019097); Dr Corfield is
supported by funding from the Norwegian
South-East Regional Health Authority (Helse
Sør-Øst; grant 2021045) and the Research Council
of Norway (grant 274611) and is a member of the
MRC Integrative Epidemiology Unit at the
University of Bristol, which is supported by the
Medical Research Council and the University of
Bristol (MC_UU_00032/1); Dr Hannigan is
supported by funding from the Norwegian
South-East Regional Health Authority (Helse
Sør-Øst; grant 2022083); Dr Bulik is supported by
the US National Institute of Mental Health (grants
R01MH136149, R01MH134039, R56MH129437,
R01MH120170, R01MH124871) and the Swedish
Research Council (grant 538-2013-8864 and
2024-02450); DrWatson received support from
the Foundation of Hope; Dr Dinkler is funded by the
Swedish Society for Medical Research (SSMF, grant
PG-22-0478); Dr Chawner is funded by a Medical
Research Foundation (Fellowship
MRF-058-0015-F-CHAW) and theWellcome Trust
(Career Development Award 304028/Z/23/Z); and
Dr Havdahl is supported by the Research Council of
Norway (grant 336085), the South-Eastern Norway
Regional Health Authority (grant 2020022), and
the European Union’s Horizon Europe Research and
Innovation program (FAMILY grant 101057529).
The genotype data was provided by the HARVEST
collaboration (supported by the Research Council
of Norway grant 229624, the NORMENT Centre
(RCN grant 223273, South-Eastern Norway Regional
Health Authority [SENRHA], and Stiftelsen Kristian
Gerhard Jebsen) in collaboration with deCODE
Genetics and the Center for Diabetes Research at
the University of Bergen (funded by the ERC AdG
project SELECTionPREDISPOSED, Stiftelsen
Kristian Gerhard Jebsen, Trond Mohn Foundation,
the Research Council of Norway, the Novo Nordisk
Foundation, the University of Bergen, and the
Western Norway Regional Health Authority).